CUTTING EDGE CARDIOVASCULAR RESEARCH
Nora Eccles Harrison Cardiovascular Research and Training Institute (CVRTI) delivers cutting-edge cell-to-bedside research and education of cardiovascular disease, which is one of the leading causes of death worldwide. At the CVRTI, we are both developing new insights into the biology of heart muscle cells, and developing novel therapeutics for patients with heart failure and cardiac arrhythmias such as sudden cardiac death.
Located at the University of Utah, the CVRTI nucleates a campus wide, multidisciplinary team of fourteen individual investigator laboratories who are both scientists and physician scientists. The research of the laboratories spans from basic muscle biology and channel electrophysiology to metabolism and genetics. Founded in 1969, the CVRTI is one of the oldest cardiovascular institutes in the country, and its research has already impacted clinical care from development of the first artificial heart, to the genetic basis of long QT arrhythmias, to using electricity to map heart dimensions for arrhythmia ablation, to myocardial recovery.

CVRTI Seminar Series
Thursday, April 10, 2025
12:00 PM – 1:00 PM MT

Visualizing Autonomic Control of Cardiac Electrophysiology
Crystal M Ripplinger, PhD, FHRS, FAHA
Professor and Vice Chair for Research and Administration
Department of Pharmacology
University of California Davis School of Medicine
Join us in person only at
Eccles Health Sciences Education Building, EHSEB, Bldg. 575, Room 2680, 25 S. 2000 E. (Lunch Provided)
or contact Nuria.Anderson@utah.edu for Zoom link.
Heart failure with reduced ejection fraction (HFrEF) has historically been a challenging condition to treat, but scientific advancements and technological innovations are transforming the way it is managed. Current research is shedding light on novel treatment options, emerging therapies, and breakthroughs in pharmaceutical development that offer hope for patients. Understanding these advancements can help improve outcomes and quality of life for those with HFrEF.
